<?xml version="1.0" encoding="UTF-8" standalone="yes"?>
<STUDY_SET>
    <STUDY alias="DRP003311" center_name="NCCRI" accession="DRP003311">
        <IDENTIFIERS>
            <PRIMARY_ID label="BioProject ID">PRJDB5213</PRIMARY_ID>
        </IDENTIFIERS>
        <DESCRIPTOR>
            <STUDY_TITLE>ChIP-seq for PolQ</STUDY_TITLE>
            <STUDY_TYPE existing_study_type="Other"/>
            <STUDY_ABSTRACT>ChIP-sequence analyses were performed under DNA replication stress in human colorectal cancer cell line HCT116 that was transfected Flag-tagged PolQ expression vector. DNA replication stress was caused by 1 mM hydroxylurea treatment for 2 hr in S-phase HCT116 cells. ChIP was performed using anti-Rad51, -Flag, and -PolH antibodies as well as non-immune IgG.</STUDY_ABSTRACT>
            <CENTER_PROJECT_NAME>ChIP-seq for PolQ</CENTER_PROJECT_NAME>
            <RELATED_STUDIES>
                <RELATED_STUDY>
                    <RELATED_LINK>
                        <DB>bioproject</DB>
                        <ID>PRJDB5213</ID>
                        <LABEL>PRJDB5213</LABEL>
                    </RELATED_LINK>
                    <IS_PRIMARY>true</IS_PRIMARY>
                </RELATED_STUDY>
            </RELATED_STUDIES>
            <STUDY_DESCRIPTION>ChIP-sequence analyses were performed under DNA replication stress in human colorectal cancer cell line HCT116 that was transfected Flag-tagged PolQ expression vector. DNA replication stress was caused by 1 mM hydroxylurea treatment for 2 hr in S-phase HCT116 cells. ChIP was performed using anti-Rad51, -Flag, and -PolH antibodies as well as non-immune IgG.</STUDY_DESCRIPTION>
        </DESCRIPTOR>
    </STUDY>
</STUDY_SET>
