<?xml version="1.0" encoding="UTF-8"?>
<STUDY_SET xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
  <STUDY center_name="GEO" alias="GSE159353" accession="SRP286990">
    <IDENTIFIERS>
      <PRIMARY_ID>SRP286990</PRIMARY_ID>
      <EXTERNAL_ID namespace="BioProject" label="primary">PRJNA668532</EXTERNAL_ID>
      <EXTERNAL_ID namespace="GEO">GSE159353</EXTERNAL_ID>
    </IDENTIFIERS>
    <DESCRIPTOR>
      <STUDY_TITLE>EphA2-SE-KO RNA-seq</STUDY_TITLE>
      <STUDY_TYPE existing_study_type="Transcriptome Analysis"/>
      <STUDY_ABSTRACT>We used Crispr/Cas9 technology to establish a homozygous clone of EphA2-SE deletion in tumor cells. Wild-type cells (WT) and homozygous cloned cells (EphA2-SE-/-) were selected for high-throughput data detection. Overall design: mRNA profiles of WT and EphA2-SE-/- cells.</STUDY_ABSTRACT>
      <CENTER_PROJECT_NAME>GSE159353</CENTER_PROJECT_NAME>
    </DESCRIPTOR>
    <STUDY_LINKS>
      <STUDY_LINK>
        <XREF_LINK>
          <DB>pubmed</DB>
          <ID>33712565</ID>
        </XREF_LINK>
      </STUDY_LINK>
    </STUDY_LINKS>
  </STUDY>
</STUDY_SET>
