<?xml version="1.0" encoding="UTF-8"?>
<STUDY_SET xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
  <STUDY center_name="GEO" alias="GSE186123" accession="SRP342099">
    <IDENTIFIERS>
      <PRIMARY_ID>SRP342099</PRIMARY_ID>
      <EXTERNAL_ID namespace="BioProject" label="primary">PRJNA772698</EXTERNAL_ID>
      <EXTERNAL_ID namespace="GEO">GSE186123</EXTERNAL_ID>
    </IDENTIFIERS>
    <DESCRIPTOR>
      <STUDY_TITLE>Regulation of Chromatin Accessibility by hypoxia and HIF_2</STUDY_TITLE>
      <STUDY_TYPE existing_study_type="Other"/>
      <STUDY_ABSTRACT>ATAC-seq was performed to investigate changes in chromatin accessibility in repsonse to HIF stabilisation via VH298 Overall design: HIF 1/2-a stabilisation was induced independent of dioxygenase inhibition by treating HeLa cells with 100 µM VH298 for 24 hs. DMSO treated cells were used as control. ATAC-seq was used to determine chromatin accessibility changes.</STUDY_ABSTRACT>
      <CENTER_PROJECT_NAME>GSE186123</CENTER_PROJECT_NAME>
    </DESCRIPTOR>
    <STUDY_LINKS>
      <STUDY_LINK>
        <XREF_LINK>
          <DB>pubmed</DB>
          <ID>35258521</ID>
        </XREF_LINK>
      </STUDY_LINK>
    </STUDY_LINKS>
  </STUDY>
</STUDY_SET>
