<?xml version="1.0" encoding="UTF-8"?>
<STUDY_SET xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
  <STUDY center_name="GEO" alias="GSE106623" accession="SRP124659">
    <IDENTIFIERS>
      <PRIMARY_ID>SRP124659</PRIMARY_ID>
      <EXTERNAL_ID namespace="BioProject" label="primary">PRJNA417537</EXTERNAL_ID>
      <EXTERNAL_ID namespace="GEO">GSE106623</EXTERNAL_ID>
    </IDENTIFIERS>
    <DESCRIPTOR>
      <STUDY_TITLE>Chromatin accessibility of GIST48, GIST882 and GIST-T1 cells with ETV1 or FOXF1 knockdown</STUDY_TITLE>
      <STUDY_TYPE existing_study_type="Other"/>
      <STUDY_ABSTRACT>ATAC profiles were generated by deep sequencing using Illumina HiSeq. Overall design: To directly examine whether FOXF1 can modulate chromatin accessibility and help guide binding of other transcription factors, we performed assay for transposase-accessible chromatin coupled with next generation sequencing (ATAC-seq) in GIST48, GIST882, and GIST-T1 cells</STUDY_ABSTRACT>
      <CENTER_PROJECT_NAME>GSE106623</CENTER_PROJECT_NAME>
    </DESCRIPTOR>
    <STUDY_LINKS>
      <STUDY_LINK>
        <XREF_LINK>
          <DB>pubmed</DB>
          <ID>29162563</ID>
        </XREF_LINK>
      </STUDY_LINK>
    </STUDY_LINKS>
    <STUDY_ATTRIBUTES>
      <STUDY_ATTRIBUTE>
        <TAG>parent_bioproject</TAG>
        <VALUE>PRJNA417469</VALUE>
      </STUDY_ATTRIBUTE>
    </STUDY_ATTRIBUTES>
  </STUDY>
</STUDY_SET>
