<?xml version="1.0" encoding="UTF-8"?>
<STUDY_SET xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
  <STUDY center_name="GEO" alias="GSE114015" accession="SRP144498">
    <IDENTIFIERS>
      <PRIMARY_ID>SRP144498</PRIMARY_ID>
      <EXTERNAL_ID namespace="BioProject" label="primary">PRJNA454880</EXTERNAL_ID>
      <EXTERNAL_ID namespace="GEO">GSE114015</EXTERNAL_ID>
    </IDENTIFIERS>
    <DESCRIPTOR>
      <STUDY_TITLE>RNA sequencing data of Wild Type and Fmr1 KO hippocampal neuron</STUDY_TITLE>
      <STUDY_TYPE existing_study_type="Transcriptome Analysis"/>
      <STUDY_ABSTRACT>Fragile X syndrome (FXS), caused by mutations in fragile X mental retardation 1 gene (FMR1), is a prevailing genetic disorder of intellectual disability and autism. Analysis of transcriptome outcome (differentially expressed genes between WT and Fmr1 KO hippocampal neuron) associated with FXS reveal promising value of gene signature-based computation in repurposing drugs for potential practical treatment. Overall design: Primary DIV (Days in vitro) 14 Hippocampal neuron mRNA profiles of Wild Type (WT) and Fmr1 KO mice were generated in triplicate</STUDY_ABSTRACT>
      <CENTER_PROJECT_NAME>GSE114015</CENTER_PROJECT_NAME>
    </DESCRIPTOR>
    <STUDY_LINKS>
      <STUDY_LINK>
        <XREF_LINK>
          <DB>pubmed</DB>
          <ID>32179850</ID>
        </XREF_LINK>
      </STUDY_LINK>
    </STUDY_LINKS>
  </STUDY>
</STUDY_SET>
