SRP173890 PRJNA510746 GSE124005 Genomic instability in the naturally and prematurely aged myocardium [RNA-Seq] Our data demonstrate that genomic instability does not evidently contribute to naturally aging of the mouse heart and support the contention that the endogenous DNA repair machinery is remarkably active to maintain genomic integrity in cardiac cells throughout life. Overall design: Investigation of variants in RNAseq for four different accelerated aging mouse models as well as natural aging at 3 different time points, each with 4 replicates. GSE124005 pubmed 34465617 parent_bioproject PRJNA510744