<?xml version="1.0" encoding="UTF-8"?>
<STUDY_SET xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
  <STUDY center_name="GEO" alias="GSE133967" accession="SRP213686">
    <IDENTIFIERS>
      <PRIMARY_ID>SRP213686</PRIMARY_ID>
      <EXTERNAL_ID namespace="BioProject" label="primary">PRJNA553220</EXTERNAL_ID>
      <EXTERNAL_ID namespace="GEO">GSE133967</EXTERNAL_ID>
    </IDENTIFIERS>
    <DESCRIPTOR>
      <STUDY_TITLE>Genome-wide ChIP-Seq from GSK-J4 or siKDM6B-treated lung adenocarcinoma cell line A549</STUDY_TITLE>
      <STUDY_TYPE existing_study_type="Other"/>
      <STUDY_ABSTRACT>Purpose: The goals of our study were to identify downstream pathway regulated by GSK-J4 in lung adenocarcinoma Methods: Chromatin isolated from GSK-J4- or siKDM6B-treated lung adenocarcinoma with proper controls was used to generate ChIP-Seq libraries Overall design: ChIP-Seq with indicated antibodies (H3K27me3 or H3K4me3) of human lung adenocarcinoma cell lines with various conditions</STUDY_ABSTRACT>
      <CENTER_PROJECT_NAME>GSE133967</CENTER_PROJECT_NAME>
    </DESCRIPTOR>
    <STUDY_ATTRIBUTES>
      <STUDY_ATTRIBUTE>
        <TAG>parent_bioproject</TAG>
        <VALUE>PRJNA553219</VALUE>
      </STUDY_ATTRIBUTE>
    </STUDY_ATTRIBUTES>
  </STUDY>
</STUDY_SET>
